Ian Robinson
Member
- Relationship to Diabetes
- Type 2
- Pronouns
- He/Him
My friend who has become a diabetes expert has said I must not eat any cheese absolutely none. Read on the traffic light food it is in the green section, full fat cheese, hmm
My 'anti D' regime is probably high cheese, at least by todays standards - I am sure some 'health experts' would come over all faint if they looked at my fridges and freezer contents.My friend who has become a diabetes expert has said I must not eat any cheese absolutely none. Read on the traffic light food it is in the green section, full fat cheese, hmm
Because I got diabetes lolCheese is OK - protein & calcium, low in carbs and good vitamins.
It's main drawbacks can be salt content, saturated fat and calories.
Pair it with other good stuff and it's fine - I use feta with avocado and tomatoes to make a nice salad.
How did you friend become a 'diabetes expert?'
Ask him when he got his degree in endocrinologyBecause I got diabetes lol
It depends on the individual as to how your body copes with carbs, some people would get high levels if they have more than a certain amount at any one meal and therefore tend to have them more evenly during the day with perhaps slightly less at breakfast as people are often more sensitive to carbs in the morning.Do you lot spread your carbs out equally between you meals?
I don't have carbs for breakfast now I just have two hard boiled eggs and coffee, it lasts me well until lunch. So I have the carbs for lunch and dinner
The DiRECT 'study' was restricted to T2s under 6 years, it couldn't have shown benefits for 10 and 25 year duration T2s. In addition DiRECT specifically made no record of complications even over its 5 year reporting period - an egregious error since the impact of any management system on T2 complications is one of the most basic things we want to know. Lean and Taylor quoted the UKPDS and weakly assumed any improvement in control would lead to reduction in the risk of complications. Of course the UKPDS has been exposed as an example of classic 'bad science' by Ben Goldacre in his book Bad Science.
Ben Goldacre's criticism of UKPDS was that it used a particular trick. Studied four variables at once and then conflated the results and averaged them out. UKPDS studied laser treatment for retinopathy, cvd, neuropathy and kidney issues ( from memory). It found Good Control reduced the risk of complications by 22%. Good news and a basic glimmer of hope although it does suggest even with Good Control a T2 retains 78% of the risk of complications. Ben Goldacre dived into the detail and found that in UKPDS Good Control reduced the risk of laser treatment for retinopathy by 83% but for the other risks the reduction something like 2,1 and 2 %. Add the four figures together and get 88, divide by 4 and get 22% reduction in risk of complications with Good Control. The best news we've got but basically averaging results like that is a bit of a fiddle as Ben Goldacre pointed out. And his main aim was to call out that technique of massaging results not the UKPDS in particular. It's widely quoted because we've got precious little else to cheer us up.Yes, I saw that.
The book by Goldacre that mentions this study was Bad Pharma, not Bad Science. And its criticisms were mainly about the scrutiny required on large trials to ensure the conclusions are good, so the comment above is very misleading as 'Bad Science' is about quacks who sell vitamin supplements and homeopathy.
The study was highly influential and is still cited by the ADA, NICE, and WHO. And it was the study that led to the use of Metformin as a first line treatment.
I try and spread them evenly across 3 meals. I rarely snack these days so the only extra carbs I have is a small amount of milk in tea or coffee.Do you lot spread your carbs out equally between you meals?
I don't have carbs for breakfast now I just have two hard boiled eggs and coffee, it lasts me well until lunch. So I have the carbs for lunch and dinner
How did they define "good control"? If it was purely HbA1c then we know that can be flawed because highs and lows can cancel each other out and result in a good HbA1c but I believe it is glucose variability/instability which is most likely to cause complications. I think with increased use of CGMs, better diabetes management can be achieved with less variability and perhaps result in improving those percentages of complication risk.It found Good Control reduced the risk of complications by 22%. Good news and a basic glimmer of hope although it does suggest even with Good Control a T2 retains 78% of the risk of complications.
I believe good control was < 54 as this was the average of the ‘intensive’ therapy group. (They had two groups, intensive and non-intensive)How did they define "good control"? If it was purely HbA1c then we know that can be flawed because highs and lows can cancel each other out and result in a good HbA1c but I believe it is glucose variability/instability which is most likely to cause complications. I think with increased use of CGMs, better diabetes management can be achieved with less variability and perhaps result in improving those percentages of complication risk.