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Advice regarding managing Type2 without medication.

beating_my_betes said:
Why would that be the case?
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Because the pancreas can't handle the glucose from the high carb load... I'd guess. Therefore sticking to a low carb diet can keep levels in check.
 
harbottle said:
Because the pancreas can't handle the glucose from the high carb load... I'd guess. Therefore sticking to a low carb diet can keep levels in check.
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What do you mean when you say that the pancreas can't handle the glucose? Also, I'm curious why you seem to be suggesting this would become worse over time.
 
beating_my_betes said:
What do you mean when you say that the pancreas can't handle the glucose? Also, I'm curious why you seem to be suggesting this would become worse over time.
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I'm not trying to answer for Harbottle. However, I feel the need to express concern that you seem to be encouraging people with Type 2 (both in this case and generally) to eat more carbs than is usually medically recommended. What's your motivation for doing this?

I also notice that, at the bottom of your profile, you advise "eating all the carbs": which carbs is it that you're suggesting people eat "all" of - and again, what's your motivation for doing that? At the top of this forum it states that, "Members are not qualified to give medical advice" and yet that seems to be what you're trying to do - and advice that appears to run counter to normal medical practice.
 
beating_my_betes said:
I'm not encouraging anyone to do anything. And this thread is not about me.

Perhaps you might answer the question you quoted.
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I have no idea what the answer is to your question: I don't pretend to any specialist knowledge about the pancreas and I prefer to follow standard medical guidance with regards to my diabetes.
 
I'm not encouraging anyone to do anything, nor am I offering medical advice. Were I to offer advice, it would fall completely in line with normal medical practice and government guidelines for both general health and diabetes.
 
beating_my_betes said:
What do you mean when you say that the pancreas can't handle the glucose? Also, I'm curious why you seem to be suggesting this would become worse over time.
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Because most people with type 2 diabetes don’t produce enough insulin due to beta cell dysfunction. Which is why blood sugar runs high.

I’m not ‘suggesting’ anything - type 2 diabetes is generally considered a progressive condition that worsens over time, especially if not controlled well.
 
harbottle said:
Because most people with type 2 diabetes don’t produce enough insulin due to beta cell dysfunction. Which is why blood sugar runs high.

I’m not ‘suggesting’ anything - type 2 diabetes is generally considered a progressive condition that worsens over time, especially if not controlled well.
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Where would you place insulin resistance in this equation?
 
When I got hold of test results from years back I saw that my blood glucose levels had ben high for years, and when it was getting really bad the GP just cancelled the test rather than do anything about it - ten years on and I'm being told I'm a very bad diabetic.
Having been able to return to normal numbers just by reducing my carb intake, I suspect that leaving me unaware caused the problem to become more entrenched.
By continuing to eat low carb I can now cope with carbs better than before, but if I were to 'eat normally' I would be retracing my way towards a high HbA1c.
With a BSc in Biology and an interest in Human evolution I have to ask if 'normal' eating is what the supermarket sells or how we evolved over the last million years.
 
There are plenty of papers showing how insulin resistance and impaired glucose tolerance leads to diabetes.
 
beating_my_betes said:
I'm not encouraging anyone to do anything, nor am I offering medical advice. Were I to offer advice, it would fall completely in line with normal medical practice and government guidelines for both general health and diabetes.
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... and yet, in your profile, you exhort people to, "Improve insulin-sensitivity by eating all the carbs!!!". Regardless of whatever it's supposed to mean, it reads like medical advice - which could mislead new members of the forum.
 
beating_my_betes said:
But wouldn't you agree that in many cases it's possible to reverse insulin resistance?
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I'm curious about how you measure this thing called insulin resistance.

To my simple mind, its easy. When a a blood glucose levels is measured an insulin level should also be measured. the ratio should tell you something about the underlying cause of high blood glucose, .

Anybody know why this is not done?
 
CliffH said:
... and yet, in your profile, you exhort people to, "Improve insulin-sensitivity by eating all the carbs!!!". Regardless of whatever it's supposed to mean, it reads like medical advice - which could mislead new members of the forum.
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Would "Improve insulin-sensitivity by decreasing the amount of carbs" be considered medical advice?
 
beating_my_betes said:
Would "Improve insulin-sensitivity by decreasing the amount of carbs" be considered medical advice?
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I don't know: I'm not feeling well enough atm to process the syntax of the sentence :(. In any case, though, I have no specialist knowledge about "insulin-sensitivity".
 
Docb said:
I'm curious about how you measure this thing called insulin resistance.

To my simple mind, its easy. When a a blood glucose levels is measured an insulin level should also be measured. the ratio should tell you something about the underlying cause of high blood glucose, .

Anybody know why this is not done?
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IR can be measured with a Homa-IR test. However - and this is just my unqualified opinion - very specific types of home testing, in certain contexts might work as a proxy.
 
CliffH said:
I don't know: I'm not feeling well enough atm to process the syntax of the sentence :(. In any case, though, I have no specialist knowledge about "insulin-sensitivity".
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Sorry you're not feeling well.
 
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Docb said:
Anybody know why this is not done?
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cPeptide tests can measure the amount of homegrown insulin produced either as part of Basal Metabolic Rate (eg fasting), or stimulated by a meal or other intake of carbs.

My understanding is that in the early stages of T2 insulin resistance is often part of the picture. Sometimes an illustration of key and lock is used - the lock is rusty or ill fitting in T2 so the insulin doesn’t work effectively. In order to process dietary carbs more insulin is produced. Some T2 meds (eg gliclazide) encourage the pancreas to produce more insulin that it would naturally.

So in some cases of T2 a cPeptide might be expected to show a high level of insulin in the presence of elevated glucose - lots of insulin, but it’s not working well.

Over time in T2 insulin signalling can be impaired and beta cell function can be compromised. So insulin production can fall to low levels, and exogenous insulin can be required.

Various things can improve insulin sensitivity and pancreatic function. Weight loss / visceral fat loss being one.

Some people seem to find that very low carb intake / keto can increase insulin resistance (which doesn’t seem to happen so much on moderate-low carb menus of 90-130g a day from what I see on the forum). But other forum members seem to get on fine at very low carb intakes, as measured by HbA1c - so very low carb seems to suit some cases/individuals.
 
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I sometimes wonder if the "insulin resistance" mentioned as being caused by a low carb diet is in reality just protein release. For me on a low carb way of eating, I have to inject insulin up front for the few carbs I eat and then I have to inject more insulin 2+ hours later for the protein release. This may increase my overall insulin requirements slightly but I don't see it as insulin resistance and it hasn't increased over time. I still use a 1:10 ratio for carbs that I used at diagnosis but I have to inject extra later for protein unless I exercise the protein release off. Libre allows me to see the protein release whereas before CGM people would just have seen their levels high before the next meal when they finger pricked and assume their normal insulin ratio was not working and they needed more and were becoming more insulin resistant.

I personally see insulin resistance as a sliding scale which pretty well everyone has to some extent and it varies from person to person and at different times of the day and how high or low your BG levels are at at any particular time. High BG levels seem to generate more resistance and low BG levels seem to make you more sensitive. That is my observation as a low carbing Type 1.
 
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