newly diagnosed - type 3c?

[rebrascora]

I was just referring to the status on this forum

If you click on your username at the top right hand side of this page next to where you see the alerts, your profile page will come up and you should just be able to select a different option for Type.

 

[angelaofthenorth]

If you click on your username at the top right hand side of this page next to where you see the alerts, your profile page will come up and you should just be able to select a different option for Type.

thanks!

 

[Proud to be erratic]

Thanks so much. Why is ketosis what people aim for in those diets then? if its so dangerous?

A keto diet, or more accurately ketogenic diet, is something some people find helpful. Essentially it is ultra low carb, typically 70-75% fat, 20%protein and 10-5 % carbs and intended to make your body create glucose from metabolising fats and proteins, rather than from negligible carbs.

Our bodies depend on glucose; our brains only use glucose and will force the metabolism of fats and proteins into carbs at the expense of denying glucose to other organs. One of the challenges is deriving the equivalent of a carb count for fats and proteins. We can glean a sufficiently robust carb content of any one food type, from cheese (nil) to cornflour (100%) and know a fair bit about the glycaemic index for many different carb foodstuffs; thus how quickly, roughly, the carbs take to be digested and how long before arriving in the bloodstream. There is enough "science" in such assessments to call the process counting, rather than guessing; but we are all different and our bodies behave differently, so the science and maths has to take into account foods that don't follow the rules. Fortunately not too many foods misbehave for most of us.

But fats and proteins are far more tricky to estimate their glucose potential from, neither being a consistent insulin to glucose conversion rate for each type and varied within types of fats and proteins. I'm in favour of an easier life when possible and follow Gary Scheiner's advice in his excellent book "Think Like a Pancreas"; he says any main meal of more than 30 gms carbs will stop the possibility of fat or protein conversion adding to your carb count. Thst isn't particularly onerous, 3 meals is still well insidethe 139gms of carbs that many people feel is a more 'normal' low carb regime. I still count the carbs in snacks, such as a frothy coffee and a cookie and take a view on whether I need to bolus for that snack depending on my BG. Such snacks sit, for me, outside the 30gm carb guideline.

I have no weight problem or dietary funnies, so I eat what I want and bolus accordingly. But others do sometimes need to avoid carbs, or just feel better on low carbs and thus follow a keto(genic) (not ketosis) diet. Its worth having this in the back of your mind, since at least one forum T2 member repeatedly encourages all and sundry to go low carb, oblivious of the risk to insulin dependent Ds -T1 or T2. If you've taken insulin, your body needs enough glucose to arrive at the right time for the insulin; or hypos become not just a risk, but potentially really dangerous: possible coma and even death if no-one close by to give (or get) help.

All advice or suggestions on this forum, especially mine (!) needs to be kept in context and seen as potentially interesting and useful but not dogmatically right.

 

[rebrascora]

Our bodies depend on glucose; our brains only use glucose and will force the metabolism of fats and proteins into carbs at the expense of denying glucose to other organs.

I personally consider this statement as scaremongering. I cannot begin to imagine a situation where a dietary regime will result in this situation and particularly, as diabetics, we usually have enough or a surfeit of glucose in our blood unless we have used too much insulin..... which yes, happens from time to time even with people on a normal or high carb diet, but since the OP is not on insulin then it is not really relevant and indeed the whole of your response is very much from an insulin user's perspective, which is different from the OP's situation at present. The enquiry was about ketosis as a result of a ketogenic diet and how that differs from DKA or at least that is how I read it.

As far as following a low carb diet being more difficult with insulin use, I actually find it easier and provided that I have a CGM of some type (Libre in my case), it actually involves less calculation. My BG levels usually move more slowly so I have more time to react and adjust my insulin and I do that as a reaction to my BG levels rising or falling rather than calculated values, which means I am responding to how my body reacts on this particular day in these particular circumstances rather than how it should respond, if that makes sense. I can't remember the last time I weighted or measured something and calculated. I just inject a nominal amount of insulin for a meal (usually 2-3 units) and then adjust afterwards either with insulin or carbs (sugar surf if you like) if necessary to keep in range.
I do not advocate low carb for everyone but it doesn't have to be difficult especially with the widespread availablity of Libre now and can in my experience be easier. I can and will still have a piece of cake occasionally or a sandwich or whatever and "wing it" with bigger insulin doses, but mostly my low carb higher fat way of eating is just easier for me to manage my diabetes with insulin as well as feeling healthier and fitter and helping me to control my disordered eating..... and my brain still gets plenty of glucose as do my other organs.

 

[angelaofthenorth]

Hi All

Quick update on my situation. My GP has been taking various blood tests this last week - two readings of hba1c 105 and 118, Cholesterol 10.9. They wanted to put me on metaformin right away, but I queried type 3c because of previous pancreatitis. So they agreed to give me the test that determines type 2 or otherwise but it will be another 7 days before the results of that test come in, so meanwhile I have been prescribed gliclozide although it wont be ready for collection until saturday so not started it yet.

Does anyone have any experience of glicozide? bit concerned it will cause me to gain weight as I already have 2 stone still to lose (after previously losing 3 stone)

 

[rebrascora]

Hi All

Quick update on my situation. My GP has been taking various blood tests this last week - two readings of hba1c 105 and 118, Cholesterol 10.9. They wanted to put me on metaformin right away, but I queried type 3c because of previous pancreatitis. So they agreed to give me the test that determines type 2 or otherwise but it will be another 7 days before the results of that test come in, so meanwhile I have been prescribed gliclozide although it wont be ready for collection until saturday so not started it yet.

Does anyone have any experience of glicozide? bit concerned it will cause me to gain weight as I already have 2 stone still to lose (after previously losing 3 stone)

Hi again.
Good to hear that your doc is authorizing more tests. Was that the C-peptide test and if so, was it a blood or urine test?
That follow up HbA1c result is rather worrying. Have you made any changes to your diet to reduce carbs? It will be improtant to drink plenty of water or low calorie drinks to help flush some of that glucose out of your system.

If your pancreas is failing then it is likely that the Gliclazide will have little effect on your BG levels or trigger any weight gain. If you are unable to produce enough insulin yourself then you usually lose weight because the body cannot access the glucose in your blood stream so it starts to break down the fat and sometimes protein (muscle mass) from your body stores to give you energy instead.
Have you adjusted your diet to reduce the amount of glucose you are putting into your body and drinking plenty of water to help flush some of that excess glucose out?
Have they given you ketone testing strips (Ketostix) or did you buy some yourself? If so, are you testing for ketones.

I believe you have bought a BG meter but if they are prescribing you Gliclazide then they should also be giving you a BG meter and test strips on prescription. What sort of readings are you getting now... with your own meter or theirs? You should be testing your urine for ketones when your BG levels are mid teens or above. If you get a Ketone reading above 1 and your BG levels are high then you need urgent medical assistance so it is important to test to keep yourself safe.

@everydayupsanddowns Would you be able to split @angelaofthenorth's posts and replies into a thread of her own, so that it doesn't get mixed up, since her circumstances are a bit different from your average Type 2 and therefore replies need to be more specific.

 

[angelaofthenorth]

Thanks Barbara, I really appreciate the response. Yes the nurse has said that they will give me BG monitor so that they can monitor the readings better rather than using mine. I did actually go to A&E a week on friday because my reading was 33. They tested me for ketones which were 0.4. All my other vital signs(kidneys liver etc) were in range so they eliminated diabetic emergency and referred me for urgent attention of my GP

 

[everydayupsanddowns]

@everydayupsanddowns Would you be able to split @angelaofthenorth's posts and replies into a thread of her own, so that it doesn't get mixed up, since her circumstances are a bit different from your average Type 2 and therefore replies need to be more specific.

Done 🙂

 

[everydayupsanddowns]

I did actually go to A&E a week on friday because my reading was 33. They tested me for ketones which were 0.4. All my other vital signs(kidneys liver etc) were in range so they eliminated diabetic emergency and referred me for urgent attention of my GP

Sorry to hear you needed to take a trip to A&E, but glad everything checked out OK.

Might it be worth getting hold of some urine ketone strips from your local pharmacy? You can buy a pot of 50 for a fiver, and while they aren’t as accurate and immediate as blood strips (£2.50 each!), they would at least give you an indication of any build-up of ketones if your levels go that high again.

Hope it’s not long before you can get put on insulin or something more suitable than your current meds - those levels are pretty punishing

 

[Proud to be erratic]

Thanks Barbara, I really appreciate the response. Yes the nurse has said that they will give me BG monitor so that they can monitor the readings better rather than using mine. I did actually go to A&E a week on friday because my reading was 33. They tested me for ketones which were 0.4. All my other vital signs(kidneys liver etc) were in range so they eliminated diabetic emergency and referred me for urgent attention of my GP

It does sound, in my non-medical opinion, that you do need to be on insulin. That may sound like a worse scenario than just oral meds, but if it restores some stability to your world and also puts you in a more pleasant state daily as well as a less damaging state than consistent high BGs - so be it.

The important thing is to get the right diagnosis and thus the correct category of Diabetes attributed to you. Because about 90% of people with D are T2, this disease is considered by the rule makers (NICE) to be treatable by GPs in their Surgeries, whereas T1 is referred to Specialist Clinics, usually in Hospitals.

Some T2s don't respond sufficiently to oral meds and end up needing insulin; they remain T2 and still treated by their GP; but sometimes those T2s are re-diagnosed to T1 - which they always were - just wrongly categorised originally. They should then come under the Specialists.

Those whose diabetes is actually because their pancreas is damaged (remember that T2s are producing sufficient insulin, but their bodies are resisting the insulin that is there, they have not got a damaged panc'y) are, strictly, T3. I say strictly because, surprisingly, a high proportion of medical folks are not aware of the T3 categorisation; my own GP wasn't. Fortunately that is slightly improving. Which 'flavour' of T3 depends on what caused the pancreatic damage; sub-categories go from a-k (I think). However, no matter the 'flavour' unless the T3 D is responding satisfactorily to T2 oral medications (and it sounds as though yours is not) then it is both appropriate and sensible for your GP to refer you to the Specialist teams. They understand the many types of insulin regimes and the initial contact point is a Diabetes Specialist Nurse (DSN) - not to be confused with the diabetes nurse in a GP Surgery, who deals predominantly with T2s on oral meds. The DSN has a great deal of autonomy, but also has relatively ready access to an Endocrinologist. If a change is needed to your meds then a DSN can recommend that change to your GP who will almost always write that prescription, accepting the authority of the DSN.

That is the theory. I was discharged after surgery as T1, which I never was. I had no pancreas, but not because of any autoimmune problems preventing me from producing insulin. My GP took my written T1 at face value; my first DSN was charming and helpful, but had no idea of the wider problems of having no pancreas and didn't know about T3 or T3c. My DSN and my GP colluded behind my back to ration my test strips and I had to wrestle with that stupid decision from 2 medical folks who'd stopped thinking about the consequences of no panc'y. Fortunately my rationale overtook their rationing. I dumped my first Endocrinologist who was useless. I got myself moved to another Endo, who seemed knowledgeable and who promised, in writing, to do certain things to improve my D management. Those promises were not even started so a few months later I got myself moved to a Centre of D excellence, in a different County and almost magically things improved hugely. I was lucky. I tell you this personal tale not to alarm you, but to alert you that T3 diabetes, ie from damage to a former healthy pancreas, is not widely understood and even the Specialists sometimes don't help as much as you might need. I have the D, it is in my interest to find the right person and steer my treatment, using the Specialist's skills, knowledge and prescribing power!

Anyway, it's now a further waiting game for you. If you are not a proper T1, then persist in getting your GP to accept that you could some flavour of T3 and get you referred specifically to an Endocrinologist; you might initially be outside the everyday knowledge of a DSN. With such high BGs it seems (again in my non-medical opinion) that you are digesting what you eat and the carbs are converting to glucose and reaching your blood stream. Many T3s need digestive enzymes supplements, which in medical parlance is Pancreatic Enzyme Replacement Therapy (PERT), because their damaged panc'ys have resulted in loss of or reduction of digestive enzymes - but that may not apply to you. There are different tests to verify that, which I didn't need! But I did have a malabsorption problem which plagued me post-op for almost 2 years; not only dreadful bowel problems but unpredictable conversion of carbs into glucose, thus carb counting not well related to insulin dosing.

Gary Scheiner in his book "Think Like a Pancreas" states:
Diabetes is complicated, confusing and contradictory.
So true, before one factors in pancreatic damage! Good luck, please keep us updated.

 

[angelaofthenorth]

It does sound, in my non-medical opinion, that you do need to be on insulin. That may sound like a worse scenario than just oral meds, but if it restores some stability to your world and also puts you in a more pleasant state daily as well as a less damaging state than consistent high BGs - so be it.

The important thing is to get the right diagnosis and thus the correct category of Diabetes attributed to you. Because about 90% of people with D are T2, this disease is considered by the rule makers (NICE) to be treatable by GPs in their Surgeries, whereas T1 is referred to Specialist Clinics, usually in Hospitals.

Some T2s don't respond sufficiently to oral meds and end up needing insulin; they remain T2 and still treated by their GP; but sometimes those T2s are re-diagnosed to T1 - which they always were - just wrongly categorised originally. They should then come under the Specialists.

Those whose diabetes is actually because their pancreas is damaged (remember that T2s are producing sufficient insulin, but their bodies are resisting the insulin that is there, they have not got a damaged panc'y) are, strictly, T3. I say strictly because, surprisingly, a high proportion of medical folks are not aware of the T3 categorisation; my own GP wasn't. Fortunately that is slightly improving. Which 'flavour' of T3 depends on what caused the pancreatic damage; sub-categories go from a-k (I think). However, no matter the 'flavour' unless the T3 D is responding satisfactorily to T2 oral medications (and it sounds as though yours is not) then it is both appropriate and sensible for your GP to refer you to the Specialist teams. They understand the many types of insulin regimes and the initial contact point is a Diabetes Specialist Nurse (DSN) - not to be confused with the diabetes nurse in a GP Surgery, who deals predominantly with T2s on oral meds. The DSN has a great deal of autonomy, but also has relatively ready access to an Endocrinologist. If a change is needed to your meds then a DSN can recommend that change to your GP who will almost always write that prescription, accepting the authority of the DSN.

That is the theory. I was discharged after surgery as T1, which I never was. I had no pancreas, but not because of any autoimmune problems preventing me from producing insulin. My GP took my written T1 at face value; my first DSN was charming and helpful, but had no idea of the wider problems of having no pancreas and didn't know about T3 or T3c. My DSN and my GP colluded behind my back to ration my test strips and I had to wrestle with that stupid decision from 2 medical folks who'd stopped thinking about the consequences of no panc'y. Fortunately my rationale overtook their rationing. I dumped my first Endocrinologist who was useless. I got myself moved to another Endo, who seemed knowledgeable and who promised, in writing, to do certain things to improve my D management. Those promises were not even started so a few months later I got myself moved to a Centre of D excellence, in a different County and almost magically things improved hugely. I was lucky. I tell you this personal tale not to alarm you, but to alert you that T3 diabetes, ie from damage to a former healthy pancreas, is not widely understood and even the Specialists sometimes don't help as much as you might need. I have the D, it is in my interest to find the right person and steer my treatment, using the Specialist's skills, knowledge and prescribing power!

Anyway, it's now a further waiting game for you. If you are not a proper T1, then persist in getting your GP to accept that you could some flavour of T3 and get you referred specifically to an Endocrinologist; you might initially be outside the everyday knowledge of a DSN. With such high BGs it seems (again in my non-medical opinion) that you are digesting what you eat and the carbs are converting to glucose and reaching your blood stream. Many T3s need digestive enzymes supplements, which in medical parlance is Pancreatic Enzyme Replacement Therapy (PERT), because their damaged panc'ys have resulted in loss of or reduction of digestive enzymes - but that may not apply to you. There are different tests to verify that, which I didn't need! But I did have a malabsorption problem which plagued me post-op for almost 2 years; not only dreadful bowel problems but unpredictable conversion of carbs into glucose, thus carb counting not well related to insulin dosing.

Gary Scheiner in his book "Think Like a Pancreas" states:
Diabetes is complicated, confusing and contradictory.
So true, before one factors in pancreatic damage! Good luck, please keep us updated.

Thank you so much I appreciate you sharing this experience. Is it usual for T2 to lose weight in unexplained way as I have? I thought it was usually T2 who are overweight / struggling to lose weight. You're right its really confusing ;(

 

[angelaofthenorth]

what

A keto diet, or more accurately ketogenic diet, is something some people find helpful. Essentially it is ultra low carb, typically 70-75% fat, 20%protein and 10-5 % carbs and intended to make your body create glucose from metabolising fats and proteins, rather than from negligible carbs.

Our bodies depend on glucose; our brains only use glucose and will force the metabolism of fats and proteins into carbs at the expense of denying glucose to other organs. One of the challenges is deriving the equivalent of a carb count for fats and proteins. We can glean a sufficiently robust carb content of any one food type, from cheese (nil) to cornflour (100%) and know a fair bit about the glycaemic index for many different carb foodstuffs; thus how quickly, roughly, the carbs take to be digested and how long before arriving in the bloodstream. There is enough "science" in such assessments to call the process counting, rather than guessing; but we are all different and our bodies behave differently, so the science and maths has to take into account foods that don't follow the rules. Fortunately not too many foods misbehave for most of us.

But fats and proteins are far more tricky to estimate their glucose potential from, neither being a consistent insulin to glucose conversion rate for each type and varied within types of fats and proteins. I'm in favour of an easier life when possible and follow Gary Scheiner's advice in his excellent book "Think Like a Pancreas"; he says any main meal of more than 30 gms carbs will stop the possibility of fat or protein conversion adding to your carb count. Thst isn't particularly onerous, 3 meals is still well insidethe 139gms of carbs that many people feel is a more 'normal' low carb regime. I still count the carbs in snacks, such as a frothy coffee and a cookie and take a view on whether I need to bolus for that snack depending on my BG. Such snacks sit, for me, outside the 30gm carb guideline.

I have no weight problem or dietary funnies, so I eat what I want and bolus accordingly. But others do sometimes need to avoid carbs, or just feel better on low carbs and thus follow a keto(genic) (not ketosis) diet. Its worth having this in the back of your mind, since at least one forum T2 member repeatedly encourages all and sundry to go low carb, oblivious of the risk to insulin dependent Ds -T1 or T2. If you've taken insulin, your body needs enough glucose to arrive at the right time for the insulin; or hypos become not just a risk, but potentially really dangerous: possible coma and even death if no-one close by to give (or get) help.

All advice or suggestions on this forum, especially mine (!) needs to be kept in context and seen as potentially interesting and useful but not dogmatically right

what is bolus?

 

[harbottle]

Thank you so much I appreciate you sharing this experience. Is it usual for T2 to lose weight in unexplained way as I have? I thought it was usually T2 who are overweight / struggling to lose weight. You're right its really confusing ;(

Type 2 can cause weight loss.

 

[Proud to be erratic]

what

what is bolus?

Bolus is a generic term for faster acting insulin, taken to offset glucose in the blood from eating and also taken to correct BG levels when they higher than desired. It sits alongside basal, the generic term for slower acting insulin intended to offset glucose arising from everyday living. This latter glucose mainly, not exclusively, comes from the liver, which stores glucose reserves. It get released by various hormones triggering a message to the liver to "open its doors"; some of those messages are, in effect, routine and more-or-less hidden bodily responses and some messages from external stimulants such as stress, illness, shock, excitement. The glucose release into blood occurs for all of us; for non-Ds their pancreas just does everything necessary to keep glucose and insulin in balance.

The package of basal and bolus insulins is known as Multiple Daily Injections (MDI) - which is one form of insulin treatment for Diabetes. Some people inject a "mixed" insulin of slow and fast, once or twice daily. The treatment path is usually started by Hospital based Specialist Teams, but for some T2s it is initiated by their GP as a progression from oral meds to insulin. Generally MDI provides a much more flexible way of managing D, but as the title implies needs several insulin doses daily. Mixed insulins work well for certain people, with the advantage of only one or 2 injections daily; but is more appropriate for people with sedentary lifestyles and very predictable eating times, meals and portion sizes. Basal and bolus allows dosing to be adjusted to daily changing circumstances and is a bit more representative of what a fully working pancreas does; but still well adrift from actual pancrwatic behaviour.

In my layman's opinion, because you could be T3c from previous panc'y damage, it would seem appropriate for you to be under a hospital based team, rather than a GP. Your diabetes would normally fall outside the scope of General Practice, or at least be assumed so - until proved otherwise. The pancreas is a small organ doing many life sustaining things and needs a Specialist (Endocrinologist) to establish what is and is not working well in your pancreatic functions.

As a matter of curiosity (not necessary if you'd rather not) could you share a little more about how much diagnosis and treatment was originally provided for your pancreatitis; and how long ago?

 

[angelaofthenorth]

Bolus is a generic term for faster acting insulin, taken to offset glucose in the blood from eating and also taken to correct BG levels when they higher than desired. It sits alongside basal, the generic term for slower acting insulin intended to offset glucose arising from everyday living. This latter glucose mainly, not exclusively, comes from the liver, which stores glucose reserves. It get released by various hormones triggering a message to the liver to "open its doors"; some of those messages are, in effect, routine and more-or-less hidden bodily responses and some messages from external stimulants such as stress, illness, shock, excitement. The glucose release into blood occurs for all of us; for non-Ds their pancreas just does everything necessary to keep glucose and insulin in balance.

The package of basal and bolus insulins is known as Multiple Daily Injections (MDI) - which is one form of insulin treatment for Diabetes. Some people inject a "mixed" insulin of slow and fast, once or twice daily. The treatment path is usually started by Hospital based Specialist Teams, but for some T2s it is initiated by their GP as a progression from oral meds to insulin. Generally MDI provides a much more flexible way of managing D, but as the title implies needs several insulin doses daily. Mixed insulins work well for certain people, with the advantage of only one or 2 injections daily; but is more appropriate for people with sedentary lifestyles and very predictable eating times, meals and portion sizes. Basal and bolus allows dosing to be adjusted to daily changing circumstances and is a bit more representative of what a fully working pancreas does; but still well adrift from actual pancrwatic behaviour.

In my layman's opinion, because you could be T3c from previous panc'y damage, it would seem appropriate for you to be under a hospital based team, rather than a GP. Your diabetes would normally fall outside the scope of General Practice, or at least be assumed so - until proved otherwise. The pancreas is a small organ doing many life sustaining things and needs a Specialist (Endocrinologist) to establish what is and is not working well in your pancreatic functions.

As a matter of curiosity (not necessary if you'd rather not) could you share a little more about how much diagnosis and treatment was originally provided for your pancreatitis; and how long ago?

no problem - I was hospitalised in 2015 with acute pancreatitis and again in 2016 - no episodes since. Both times I stayed for 3 days basically for pain management because it's so horrific. Fluids as well but otherwise no particular treatment, it just needs time for the 'flare up' to pass. My pancreatitis was caused by alcoholism, (which thankfully I have managed to beat)

 

[angelaofthenorth]

Bolus is a generic term for faster acting insulin, taken to offset glucose in the blood from eating and also taken to correct BG levels when they higher than desired. It sits alongside basal, the generic term for slower acting insulin intended to offset glucose arising from everyday living. This latter glucose mainly, not exclusively, comes from the liver, which stores glucose reserves. It get released by various hormones triggering a message to the liver to "open its doors"; some of those messages are, in effect, routine and more-or-less hidden bodily responses and some messages from external stimulants such as stress, illness, shock, excitement. The glucose release into blood occurs for all of us; for non-Ds their pancreas just does everything necessary to keep glucose and insulin in balance.

The package of basal and bolus insulins is known as Multiple Daily Injections (MDI) - which is one form of insulin treatment for Diabetes. Some people inject a "mixed" insulin of slow and fast, once or twice daily. The treatment path is usually started by Hospital based Specialist Teams, but for some T2s it is initiated by their GP as a progression from oral meds to insulin. Generally MDI provides a much more flexible way of managing D, but as the title implies needs several insulin doses daily. Mixed insulins work well for certain people, with the advantage of only one or 2 injections daily; but is more appropriate for people with sedentary lifestyles and very predictable eating times, meals and portion sizes. Basal and bolus allows dosing to be adjusted to daily changing circumstances and is a bit more representative of what a fully working pancreas does; but still well adrift from actual pancrwatic behaviour.

In my layman's opinion, because you could be T3c from previous panc'y damage, it would seem appropriate for you to be under a hospital based team, rather than a GP. Your diabetes would normally fall outside the scope of General Practice, or at least be assumed so - until proved otherwise. The pancreas is a small organ doing many life sustaining things and needs a Specialist (Endocrinologist) to establish what is and is not working well in your pancreatic functions.

As a matter of curiosity (not necessary if you'd rather not) could you share a little more about how much diagnosis and treatment was originally provided for your pancreatitis; and how long ago?

Thanks to your posts I have ordered 'Think like a Pancreas' arriving tomorrow!

 

[angelaofthenorth]

Ok so I've tested negative for type 1. Not sure where that leaves me regarding type 3c. Anyway I am meeting a GP whi is apparently well up on diabetes on the 10th. Ive been asked to sort out a stool sample prior to that.
Meanwhile I am going to start on Metaformin.

 

[angelaofthenorth]

Any ideas what I should say to/ask this GP when I see him next week?

 

[Proud to be erratic]

Ok so I've tested negative for type 1. Not sure where that leaves me regarding type 3c. Anyway I am meeting a GP whi is apparently well up on diabetes on the 10th. Ive been asked to sort out a stool sample prior to that.
Meanwhile I am going to start on Metaformin.

The negative just confirms that you don't have the autoimmune conditions that would make you T1. Your original diagnosis is T2, but the question in my mind would be: do you have the normal T2 characteristics, ie producing insulin but with an unduly high natural resistance to your own insulin; or is your insulin production impaired because of previous pancreatitis?

Any ideas what I should say to/ask this GP when I see him next week?

Could it be that you are not a straightforward T2? Could you be referred to an Endocrinologist for further tests?

 

[angelaofthenorth]

I will ask the GP when I see him on the 10th about the endocrinologists. Feeling a bit scared and depressed today about it all. I need to stop googling