ust get out there and do as much exercise as you can. Preferably a mix of aerobic and resistance exercise, which is what we all should be doing.
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Resistance exercise is what I'm good at, and cardio I'm rubbish at - which is probagly a sign that it is the latter that I should be doing more of, not the former (sigh). (This is the good old puritan principle that it is what we don't want to do that is what we should be doing!!!!)
I guess I just wanted to ensure I'm not doing anything that will counter my efforts to crack my pre-T2 insulin resistance!!!
Here’s a very useful academic article, published last year—a group of academics, on behalf of the American College of Sports Medicine, summarising recent research on exercise, in general and various different types, for people with Type 2:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8802999/
Loads of handy-- and highly motivating-- information! But the most important thing is the summary in the introduction:
“During
any type of physical activity (PA), glucose uptake into active skeletal muscles increases via insulin-independent pathways. … Improvements in systemic, and possibly hepatic,
insulin sensitivity following
any PA can last from 2 to 72 h, with reductions in blood glucose closely associated with PA duration and intensity. Additionally, regular PA enhances β-cell function,
insulin sensitivity, vascular function, and gut microbiota, all of which may lead to better diabetes and health management as well as disease risk reduction.” [I’ve added the underlining and removed footnotes.]
There’s a little confusion around resistance exercise; resistance exercise does
not increase insulin resistance!
Undoing the confusion requires a good deal of explanation! But, if you’re interested—the explanation, as I understand it, is as follows:
As you probably know, most of your cells most of the time need insulin in order to take up glucose, the main fuel for cells. You can picture the cell as having a little door to let in glucose-- but the door is locked, and insulin is the key.
Muscle cells, though, have ‘glucose doors’ that can be unlocked either by insulin or by exercise. When muscle cells start contracting rhythmically, after a little while that little ‘door’ opens, and they start sucking up glucose—no insulin needed.
With moderate-intensity aerobic exercise (like brisk walking or slow running), this will tend to make your blood glucose level go down. For non-diabetics and Type 2s who are not insulin dependent (i.e. not injecting artificial insulin), this is not a problem. Your beta cells will be putting out a bit of insulin most of the time; and real, natural insulin is very fast-acting and very short-acting. So—whenever your system senses that your BG is going too low, your beta cells will just stop putting out insulin for the time being; so you won’t become hypoglycaemic.
For Type 1s (and, I think, insulin-dependent Type 2s) though: Even the so-called ‘rapid acting’ artificial insulins are slower to take effect and hang around in the system longer than natural insulin. So, if we have artificial insulin in our system, and our BG is lowish, and then we go for a long, brisk walk: our muscles will start sucking up glucose, AND we’ll have insulin in our system inappropriately making our other cells also suck up glucose—and we are likely to go hypo. So we have to manage this carefully.
With resistance exercise, or high-intensity aerobic exercise: While you’re doing it, the effect of your muscles sucking up glucose may be counteracted by another factor. When you do resistance exercise or high-intensity aerobic exercise, your body tends to start putting out cortisol (a stress hormone); and cortisol triggers your liver to release stored glucose. So, while you’re doing the exercise, your BG may go up rather than down. And this may continue for a little while after you stop the exercise; you’ll see the article mentions “transient post-exercise hyperglycemia”.
But, a little while after you’ve done your resistance exercise or HIT, your body stops pumping out cortisol, so your liver stops releasing glucose, and the effect is then the same as the post-exercise effect of moderate-intensity aerobic exercise: your muscles keep sucking up glucose for hours afterwards. The benefits of this hugely outweigh any short-term rise in BG during resistance exercise or HIT.
Again, this can be tricky for Type 1s: with resistance exercise or HIT, we have to think about how to manage any hyperglycaemia during or shortly after the exercise without increasing our risk of hypoglycaemia in the following hours! But Type 2s who are not on any medication are not at any risk of hypoglycaemia, so you don’t have to worry about this.
I hope this helps, and all best wishes!