The trouble with having diabetes is this - your bodily cells can't get hold of what you are eating, and they need to get hold of it in order to function. The body converts food to glucose, and it's glucose the cells need to stay alive and function.
It works kind of like this.
The cell doors are locked so the glucose can't get in.
The brain tells the pancreas 'Here comes some food - we need to open the cell doors, so send the keys along please'
The pancreas responds by sending along some insulin.
Insulin is the key that opens the cell doors.
When you get Type 1, the bit of the pancreas that produces the insulin has died, so no insulin is available, so we have to inject it instead.
When you get Type 2, usually something else goes wrong whereby the pancreas is still doing it's bit by sending along the insulin, but the malfunction is stopping it working. If you like, has it's foot against all the cell doors to stop them opening, even when they are unlocked. This is called Insulin Resistance.
Whatever, the cell doors stay locked so the cells stay hungry.
And that's the message you get - I'm hungry! Please feed me! So you do - but it only improves things for a short time and then the cells get hungry again. And you are hungry again.
Meanwhile, because your food is still being converted to glucose but it can't get into the cells, the glucose stays in your blood and it circulates round the body till it eventually gets to your kidneys from where it passes into your urine and you pee it out. But the kidneys weren't actually meant to deal with so much glucose. Basically they act like a sieve, and the glucose molecules are too big to go through the holes, but eventually they make the holes bigger and they do get through. Because the holes get bigger, then any number of other things that shouldn't get into your kidneys also can get in, and kidney damage results.
Also, the glucose in your blood gets into your liver. This is fine under normal circumstances because the liver has this nice little mechanism where it converts it to Glycogen, which is your body's emergency supply of energy for when you need it in desperation (to run away from a murderer or whatever) But it can't store very much Glycogen so when it's full, it stores what's left over as BODY FAT. Especially the sort that gets round your internal organs. Not good.
So .... the net result is, uncontrolled diabetes makes you feel hungry even when you've had more than enough to eat.
The body converts carbohydrates to glucose much quicker (2 times) than it converts protein or fat (9 times) but if you continue to eat loads of carb, all you do is keep overloading your blood with glucose in trainloads.
But if you slow the process down, by eating less carb and more protein and fat - then your body should have a chance to catch up a bit.
Plus, protein keeps you fuller for longer anyway (with or without diabetes) - so you win all round.
A T2 friend of mine tried to cut out LORS of carb straight away. This was a mistake for her, because it made her feel worse than she started off. Do it a week at a time or something - this week, I'll cut 10%, next week another 10%.
Once you get it in control it may be possible to add some of the carbs back in, but it's unlikely you'll want them all back, by then.
And yes - you really DO need to get a meter whatever your flipping nurse or doctor says. How will you know what's OK for you to eat and what isn't so good, if you don't?
Finally we are all different - LeeLee may be able to eat this or that, Pav may be able to eat different things to LeeLee and be OK. The only way to find out what's good or bad for you, is to TEST.