I had a total pancreatectomy in Feb 20, making me instantly as if T1 (but without the autoimmune condition). My understanding (I think from Gary Scheiner's book "Think Like a Pancreas") is that our brain has no direct pathway to communicate with the liver. So when our brain detects that our BG is inappropriately low it sends a message to the pancreas, which releases the hormone Glucagon that tells the liver to open its glucose store. But that pathway can't happen for me.
At first I did not get DP or FOTF. Some 2 years in this started and I asked an Endocrinologist (from a Hospital piloting pancreatic Islet transplants) how could DP occur in me? Her comment was they weren't absolutely certain, but our brains find alternative routes and possibly my cortisol or adrenaline glands were being triggered by the brain, instead.
Subsequently I have routinely, for almost 2 years, taken upstairs a mini snack of cheese and ham slices rolled together (now findable in Tesco, Aldi and Lidl as "rollitos"; I make my own). If I wake in the small hours, or before my alarm, I eat a couple of these bites-sized indulgences and do NOT experience DP or FOTF. That small release of protein works for me in convincing my body that I'm not starving, don't need help and the quantity of protein is not big enough to eventually convert into glucose in the absence of eating an equivalent carb snack.
So
@harbottle, if you can explain a physiological pathway for this I would genuinely be interested to know.
@Beancounter298, you might fairly conclude much of our metabolic processes are not simple, for T1, T2 or T3c. Also in Gary Scheiners book he states Diabetes is Complicated, Confusing and Contradictory. That is so true.