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- Relationship to Diabetes
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A study published in the journal Nature Communications has pinpointed a number of areas of the human genome that may help explain the neonatal origins of chronic immune and inflammatory diseases of later life, including type 1 diabetes, rheumatoid arthritis and celiac disease.
The research, led by scientists at the Cambridge Baker Systems Genomics Initiative, identified several genes that appear to drive disease risk at birth, and which could be targeted for therapeutic intervention to stop these diseases in their tracks, well before symptoms occur.
Dr Michael Inouye, Munz Chair of Cardiovascular Prediction and Prevention at the Baker Institute and Principal Researcher at Cambridge University, said chronic immune and inflammatory diseases of adulthood often originated in early childhood, with an individual's genetic make-up causing changes to the function of different genes involved in disease.
The research, led by scientists at the Cambridge Baker Systems Genomics Initiative, identified several genes that appear to drive disease risk at birth, and which could be targeted for therapeutic intervention to stop these diseases in their tracks, well before symptoms occur.
Dr Michael Inouye, Munz Chair of Cardiovascular Prediction and Prevention at the Baker Institute and Principal Researcher at Cambridge University, said chronic immune and inflammatory diseases of adulthood often originated in early childhood, with an individual's genetic make-up causing changes to the function of different genes involved in disease.
Gene variations at birth reveal origins of inflammation and immune disease
A study has pinpointed a number of areas of the human genome that may help explain the neonatal origins of chronic immune and inflammatory diseases of later life, including type 1 diabetes, rheumatoid arthritis and celiac disease.
www.sciencedaily.com
