Eddy Edson
Well-Known Member
- Relationship to Diabetes
- Type 2
Diabetes review today, and HbA1c down to 5.2% = 33 mmol/mol, after being stuck at 5.3% since Jan 2020. (And sub 40 since Sep 2018.)
BFD, of course, but it's comforting to see no deterioration with advancing age so far!
Also with pretty big increase in carbs from things like noodles and rice paper. Non-diabetics and in fact (according to the evidence) generally T2D's with reasonable control don't see rises in BG in lock-step with increasing carbs. Preventing that is something that your metabolism is designed for, and it generally works well if you're not hugely insulin deficient. Your BG plateaus at some point, and there are studies which appear to show that T2D's with reasonable HbA1c's need to go full-on keto before they see a very significant impact on BG from changing carb intake.
Increasing carbs doesn't cause T2D, and if your metabolism is doing a halfway reasonable job, neither will it cause your BG control to explode.
So a lot of people should stop stressing so much about carbs, and stress more about CV issues, which are generally more important if your BG is reasonably under control.
EDIT: An interesting little study from a few months ago: https://pubmed.ncbi.nlm.nih.gov/35560850/
Summary: This study randomised people with T2D to receive five different doses of carbohydrate from 10% to 30% of calories in random order to see what effect it had on their blood glucose.
Results: Twelve participants completed the study. There were no differences in 24-h and post-prandial sensor glucose concentrations between the 30 and 10%kcal doses (7.4 ± 1.1 mmol L-1 vs. 7.6 ± 1.3 mmol L-1 [p = 0.28] and 8.1 ± 1.5 mmol L-1 vs. 8.5 ± 1.4 mmol L-1 [p = 0.28], respectively). In our exploratory analyses, we did not find any dose-response relationship between carbohydrate intake and glycaemia. A small amount of weight loss occurred in each treatment arm (range: 0.4-1.1 kg over the 6 days) but adjusting for these differences did not influence the primary or secondary outcomes.
Conclusions: Modest changes in dietary carbohydrate content in the absence of weight loss at the same time as keeping dietary protein intake constant do not appear to influence glucose concentrations in people with well-controlled T2D.
BFD, of course, but it's comforting to see no deterioration with advancing age so far!
Also with pretty big increase in carbs from things like noodles and rice paper. Non-diabetics and in fact (according to the evidence) generally T2D's with reasonable control don't see rises in BG in lock-step with increasing carbs. Preventing that is something that your metabolism is designed for, and it generally works well if you're not hugely insulin deficient. Your BG plateaus at some point, and there are studies which appear to show that T2D's with reasonable HbA1c's need to go full-on keto before they see a very significant impact on BG from changing carb intake.
Increasing carbs doesn't cause T2D, and if your metabolism is doing a halfway reasonable job, neither will it cause your BG control to explode.
So a lot of people should stop stressing so much about carbs, and stress more about CV issues, which are generally more important if your BG is reasonably under control.
EDIT: An interesting little study from a few months ago: https://pubmed.ncbi.nlm.nih.gov/35560850/
Summary: This study randomised people with T2D to receive five different doses of carbohydrate from 10% to 30% of calories in random order to see what effect it had on their blood glucose.
Results: Twelve participants completed the study. There were no differences in 24-h and post-prandial sensor glucose concentrations between the 30 and 10%kcal doses (7.4 ± 1.1 mmol L-1 vs. 7.6 ± 1.3 mmol L-1 [p = 0.28] and 8.1 ± 1.5 mmol L-1 vs. 8.5 ± 1.4 mmol L-1 [p = 0.28], respectively). In our exploratory analyses, we did not find any dose-response relationship between carbohydrate intake and glycaemia. A small amount of weight loss occurred in each treatment arm (range: 0.4-1.1 kg over the 6 days) but adjusting for these differences did not influence the primary or secondary outcomes.
Conclusions: Modest changes in dietary carbohydrate content in the absence of weight loss at the same time as keeping dietary protein intake constant do not appear to influence glucose concentrations in people with well-controlled T2D.
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