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Do I really need medicine?

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So why do you believe would it go higher on a low carb diet?

Low carb 101 from the master.


It seems like your OGTT, you seemed to have beaten the odds again here as well.
 
After 18 months.. not a few weeks....
 
Tell him your opinion is he should clarify his timescale, not me, if you are telling him he's inaccurate or too imprecise for you.
Tell who?
Which "him" are you referring to?
 
Not really the body is simply taking an opportunity to dump excess stored glucose out of the cells into the blood stream to use it up.
Once the excess glucose stores are used the FBG will revert to normal.
Took mine about 6 months to do this when ultra low carbing.

You must have a huge liver to store 6 months of glucose.

The liver (And muscles) stores some excess glucose as glycogen, the rest is turned into fat for long term storage.

The liver only stores a small amount of glycogen and uses it during fasting to keep blood sugar from going low or for when hormones demand a release (Fight or flight response).
After a few days of fasting you'd start using gluconeogenesis to generate glucose from fat and proteins as the glycogen has gone.
 
You must have a huge liver to store 6 months of glucose.

The liver (And muscles) stores some excess glucose as glycogen, the rest is turned into fat for long term storage.

The liver only stores a small amount of glycogen and uses it during fasting to keep blood sugar from going low or for when hormones demand a release (Fight or flight response).
After a few days of fasting you'd start using gluconeogenesis to generate glucose from fat and proteins as the glycogen has gone.
That's not how it works.. the damage caused by T2 is from glucose being forced into all sorts of organs where it shouldn't be.. diabetic retinopathy for example, glucose in the eye.
Unless everything I have read about T2 is wrong... (and no it isn't).
 
glucose in the eye

For clarity, my understanding is that it‘s the elevated glucose levels in the bloodstream putting stress on the very fine blood vessels which service the retina, rather than glucose causing damage by entering the eye itself (though changes in osmotic pressure can distort the shape causing blurred vision).

“Diabetes damages blood vessels all over the body. The damage to your eyes starts when sugar blocks the tiny blood vessels that go to your retina, causing them to leak fluid or bleed. To make up for these blocked blood vessels, your eyes then grow new blood vessels that don’t work well. These new blood vessels can leak or bleed easily.”

 
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That's not how it works.. the damage caused by T2 is from glucose being forced into all sorts of organs where it shouldn't be.. diabetic retinopathy for example, glucose in the eye.
Unless everything I have read about T2 is wrong... (and no it isn't).

I don’t understand what you are talking about. I never mentioned eyes or glucose being forced anywhere.
 
My experience is that Fasting BG goes down (but sometimes very slowly - think years) on Low Carb. Others are fortunate that it goes down much faster than it has for me.
It certainly didn't go up !
 
I don’t understand what you are talking about. I never mentioned eyes or glucose being forced anywhere.
You implied that glucose was only stored in the liver.
It goes all over the body.
Fructose is treated solely by the liver .. maybe that was what you meant?
 
For clarity, my understanding is that it‘s the elevated glucose levels in the bloodstream putting stress on the very fine blood vessels which service the retina, rather than glucose causing damage by entering the eye itself (though changes in osmotic pressure can distort the shape causing blurred vision).

“Diabetes damages blood vessels all over the body. The damage to your eyes starts when sugar blocks the tiny blood vessels that go to your retina, causing them to leak fluid or bleed. To make up for these blocked blood vessels, your eyes then grow new blood vessels that don’t work well. These new blood vessels can leak or bleed easily.”

" sugar blocks the tiny blood vessels that go to your retina,"

Sounds pretty much like sugars being forced into organs although possibly clumsily worded on my part?
 
You implied that glucose was only stored in the liver.
It goes all over the body.
Fructose is treated solely by the liver .. maybe that was what you meant?

I said no such thing.

I was talking about how we store excess glucose as glycogen (In muscles AND the liver) for use during periods of fasting and exercise. This is made from carbohydrates.

This is different to the blood glucose which is in the bloodstream - levels which are managed by the pancreas's beta/alpha cells and, if necessary, the conversion of proteins/fatty acids into glucose.

Have no idea why you mention fructose. That goes through a different mechanism and is metabolised as fat, hence why too much can be bad for you and possibly lead to NAFLD.
 
" sugar blocks the tiny blood vessels that go to your retina,"

Sounds pretty much like sugars being forced into organs although possibly clumsily worded on my part?

?

As the glucose in your bloodstream it goes wherever your blood goes.

Which is through organs.

And veins in the eyes.

And if there's too much glucose in it... it can cause damage where it goes.
 
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