It’s worth noting that Type 3c diabetes ( treated as Type 1, or at least should be) is now more common than T1. It is due to pancreatic destruction due to factors other than autoimmune factors. The commonest of these is chronic pancreatitis, which in some cases follows one or more episodes of acute pancreatitis caused by alcohol.
I’ve got chronic pancreatitis not due to alcohol, probably an association with ulcerative colitis or it’s treatment. More common in women is gallstones blocking the pancreatic duct, and surgery to treat non malignant tumours of the pancreas. I was T1 years before CP developed.
3c brings its own problems, by far the worst being the assumption by doctors that it’s T2 by reasons of age. The other is explaining how you got it, far more complex than a run of the mill T1, so vanilla T1s have it easy.
Most of you are clueless how Creon can interfere with how quick food is absorbed by the body, and indeed how variable it is. If you are wondering where all these 3c folk are, they are legion on the Pancreatitis forum, but diabetes is not discussed very much, nowhere near as much as pain and symptom relief.
So there you are; the commonest form of insulin dependent diabetes isn’t T1. It’s 3c. It’s still all replacement therapy, but with the bonus of having to replace digestive enzymes (the main job of the pancreas) as well as the secondary function of insulin production.
Now where were we up to in the discussion about T1s and T2s? Bit parochial, even exclusive. Shame on you.😉